Abstract Topics
Lymphoma Symposium

• Genetic changes
• Microenviromental factors of importance for oncogenesis
• Clinical behaviour

Abstract Topics
Bone Marrow Symposium

- Features of bone marrow involvement in Hodgkin lymphoma, aggressive B-NHL and peripheral T-cell lymphomas
- Non-neoplastic lymphoproliferations in the bone marrow (excluding immunodeficiency-related and EBV-associated lymphoproliferations)
- The role of the bone marrow microenvironment in lymphoma manifestations

David Y Mason's Award

The EAHP is opening the first call for the David Y Mason’s Award thanks to the generous contribution of the David Y Mason Foundation. This Award wants to foster the interest of young researchers for Hematopathology and help them to become active members of our Society.

Deadline: June 30, 2018

Travel Grants

In order to promote the educational and scientific development of Hematopathology and facilitate the participation of hematopathologists, residents and PhD students from developing countries, registration fee waivers and travel grants of € 500 will be made available to attend the 19th Symposium and Workshop of EAHP in Edinburgh, Scotland, 29 September – October 4, 2018.

Why Exhibit?

Bone Marrow Workshops

Make use of the EAHP 2018 congress as a perfect networking opportunity – meet your target group, expand your customer base and benefit from the active exchange of information on-site.

Preliminary Programme

Main topics:
Lymphoma & Bone Marrow Symposia

- Genetic changes
- Microenviromental factors of importance for oncogenesis
- Clinical behaviour


European Association for Haematopathology shared World Down Syndrome Day's photo. ... See MoreSee Less

What are you doing for #WDSD18 ? Everyone at North Shore ConneXions Society in North Vancouver will be wearing #LotsOfSocks to raise awareness about #Downsyndrome

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European Association for Haematopathology shared Haematologica's post. ... See MoreSee Less

In this study the Authors aimed to identify the genetic cause in pedigrees with non-syndromic familial MDS. They discovered constitutional SAMD9L mutations associated with non random patterns of clonal escape leading to loss of the mutant allele. They further demonstrate in two cases that SAMD9L-related disease can be associated with transient-7, occurring as a one time clonal event followed by somatic correction of hematopoiesis achieved by UPD7q with double wild-type SAMD9L www.haematologica.org/content/103/3/427

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